By Jim W. Burgess, Philip A. Sinclair, Christophe M. Chretien, Jonathon Boucher (auth.), Associate Professor Sukhinder Kaur Cheema (eds.)
About the Series:
Advances in Biochemistry in Heath and Disease offers state of the art discussions in state-of-the-art biochemical study, supplying fascinating advancements that influence healthcare and illness examine. Volumes within the sequence specialise in cross-disciplinary biomedical learn and view a number of subject matters in biochemistry, phone biology, molecular biology, and biomedicine.
Biochemistry of Atherosclerosis
Sukhinder Kaur Cheema
Biochemistry of Atherosclerosis examines atherosclerosis in nice aspect, targeting the chance of atherosclerosis, and the biochemical pathways concerned. It presents a breadth of information in addition to new insights right into a number of themes when it comes to atherosclerosis from major scientists around the globe who're on the leading edge of atherosclerosis study. Biochemistry of Atherosclerosis is key interpreting for biomedical and medical researchers.
- Hyperlipidaemia and Atherosclerosis
- Diabetes brought on Atherosclerosis
- Hypertension triggered Atherosclerosis
- Homocysteine Metabolism and Atherosclerosis
- Role of the Immune process in Atherosclerosis
- Role of Infectious brokers in Atherogenesis
- Dietary administration of Aherosclerosis
About the Editor:
Sukhinder Kaur Cheema is at the moment affiliate Professor of Biochemistry and a CIHR (Canadian Institutes of health and wellbeing learn) New Investigator on the Memorial collage of Newfoundland. knowledgeable in dietary biochemistry, lipid metabolism and heart problems, she is pass appointed in school of medication on the Memorial college of Newfoundland.
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Additional info for Biochemistry of Atherosclerosis
NG identified defective alleles further contribute to the structural heterogeneity in this disease. J Clin Invest 91(2): 677–683, 1993. Borysiewicz LK, Soutar AK, Evans DJ, Thompson GR, Rees AJ: Renal failure in familial lecithin: cholesterol acyltransferase deficiency. Q J Med 51: 411–426, 1982. Santamarina-Fojo S, Lambert G, Hoeg JM, Brewer HB Jr: Lecithin–cholesterol acyltransferase: role in lipoprotein metabolism, reverse cholesterol transport and atherosclerosis. Curr Opin Lipidol 11: 267–275, 2000.
LCAT transgenic mice have been created by a number of laboratories. As expected, a modest overexpression of the human LCAT gene in mice resulted in a moderate elevation of total plasma HDL-C levels and an increase in the HDL particle size. In case of the human LCAT gene being coexpressed with the human apoA-I transgene, a mouse strain showed a humanlike polymodal size distribution of HDL, modest particle size increase was observed in each individual HDL subfractions attributable to increased accumulation of CE .
J Biol Chem 270: 29916–29922, 1995. 33. Agellon LB, Zhang P, Jiang XC, Mendelsohn L, Tall AR: The CCAAT/enhancer-binding protein trans-activates the human cholesteryl ester transfer protein gene promoter. J Biol Chem 267: 22336–22339, 1992. 34. Jeoung NH, Jang WG, Nam JI, Pak YK, Park YB: Identification of retinoic acid receptor element in human cholesteryl ester transfer protein gene. Biochem Biophys Res Commun 258: 411–415, 1999. 35. Chouinard RA Jr, Luo Y, Osborne TF, Walsh A, Tall AR: Sterol regulatory element binding protein-1 activates the cholesteryl ester transfer protein gene in vivo but is not required for sterol up-regulation of gene expression.
Biochemistry of Atherosclerosis by Jim W. Burgess, Philip A. Sinclair, Christophe M. Chretien, Jonathon Boucher (auth.), Associate Professor Sukhinder Kaur Cheema (eds.)